Cardio Conditioning will be a priority. Starting with walking four minutes followed by a 30 second sprint for 5 rounds.
Stacking
Whey 20 to 60 grams per dayFish Oil 4 grams
Coconut Oil 1 tsp
MCT oil 1 tbl
COQ10 200mg
7-Keto DHEA 200mg
DHEA 50mg
Friday, March 29, 2013
Up to 214 lbs. (97 kg) I am instituting the HIIT program.
I have really fallen off since November. Time to get back into the fight. My food selection has been whatever I can get my hands on.
Cardio Conditioning will be a priority. Starting with walking four minutes followed by a 30 second sprint for 5 rounds.
Fish Oil 4 grams
Coconut Oil 1 tsp
MCT oil 1 tbl
COQ10 200mg
7-Keto DHEA 200mg
DHEA 50mg
Cardio Conditioning will be a priority. Starting with walking four minutes followed by a 30 second sprint for 5 rounds.
Fish Oil 4 grams
Coconut Oil 1 tsp
MCT oil 1 tbl
COQ10 200mg
7-Keto DHEA 200mg
DHEA 50mg
Wednesday, March 20, 2013
Sunday, March 17, 2013
BPI Sports RoxyLean The Healthy Supportive, Metabolic, Weight Management Agent!
RoxyLean 60ct. | On Sale Now at TigerFitness Online!
RoxyLean is a Medi-Biological Weight loss product that delivers immediate, fast acting fat loss results. Please do not take without prior consent of our physician. RoxyLean. The Healthy Supportive, Metabolic, Weight Management Agent! RoxyLean is based on the most popular weight loss supplement ever formulated. It is extremely strong and not for everyone. It is comprised of the exact ratio of energy-driven ingredients, performance based components, and circulatory catalysts that make it unlike any other weight loss product available in stores today. RoxyLean is THE WEIGHT MANGEMTNET< ENERGY ENHANCING FORMULA. So FAST, so LEAN, and so RIPPED. This is the product that delivers where all others fall short. When taken properly, research shows that the component agents in RoxyLean may have a remarkable and potentiating effect on weight management and lean muscle mass may occur. In all the years that companies have struggled to formulate effective versions of weight loss products, there has never been another company as progressive as BPI Sports in helping reshape the category of weight loss supplements. Never before has there been a product as targeted as RoxyLean-based on laboratory and scientific research and development, UNTIL NOW
Directions
Begin by taking 1 capsule in the morning on an empty stomach before food. If needed take an additional 1 capsule six hours later on an empty stomach. Do not exceed this dosage until you know how your body responds to this product. Due to the extreme strength of this product. Do Not Exceed more than 3 capsules per day.
Warnings
Not intended for use by persons under the age of 18. Do not consume synephrine or caffeine from other sources, including but not limited to coffee, tea, soda, an other dietary supplements or medications containing pherylephrine or caffeine. Contains Caffeine. Do not use for more than 8 weeks. Consult your physician prior to use if you are pregnant, nursing, or if you are taking medications including but not limited to: MAOI inhibitors, antidepressants, aspirin, nonsteroidal anti-inflammatory drugs or products containing phenylphrine, ephedrine, pseudoephedrine, or other stimulants. Consult your physician prior to use if you have a medical condition, including but not limited to: heart, liver, kidney or thyroid disease, psychiatric or epileptic disorders, difficulty urinating, diabetes, high blood pressure, cardiac arrhythmia, recurrent headaches, enlarges prostate or glaucoma. Discontinue 2 weeks prior to sugary or if you experience rapid heartbeat, dizziness, severe headaches or shortness of breath. This product may contain ingredients banned by certain sports organizations. User assumes all risks, liabilities or consequences respecting testing.
Beverly 7-Keto MuscLEAN Clinically Proven To Be Remarkably Effective For Fat Loss!
Get 7-Keto Musclelean at Tiger Fitness
Clinically Proven To Be Remarkably Effective For Fat Loss!
Besides being a powerful antioxidant, Green Tea is added for its lipolytic properties. In laymen's terms, it improves the usage of fat as a fuel source. Rounding out the stack is Guarana, which provides an amplifying, fat melting kick of caffeine. Beverly's MuscLEAN includes these three cofactors as well as the breakthrough micronutrient, 7- Keto in full potency. Take three 7-Keto MuscLEAN capsules, twice daily and you'll get the full research-proven 200 mg. dose. 7-Keto MuscLEAN will not just help you lose weight, but your weight loss will come from fat alone, not from water weight or loss of lean muscle tissue.
Proof Two randomized, double-blind, placebo-controlled, clinical studies demonstrated amazing results from a 200-mg. dose of 7-Keto. In the first 7-Keto was responsible for three times more weight loss versus placebo, with the weight loss due solely to fat reduction, not water or muscle loss. The second study confirmed these findings by testing two groups with similar diet and exercise programs. The 7-Keto group showed a dramatic improvement in body composition, i.e. fat loss/ muscle retention, in comparison to the placebo group, which reduced weight slightly due to muscle, water, and fat loss in similar amounts.
Note: Both research groups used 200 mg per day of 7-Keto in divided doses. While most products on the commercial market contain half this amount, this is the exact dosage contained in two servings of Beverly's MuscLEAN. If you want to get the research proven results, you should use the research proven amount. 7-Keto has become the world's #1 Fatloss Agent and now it's available from the world's #1 supplement company. Beverly International's 7-Keto MuscLEAN is safe, natural, and best of all guaranteed 100% effective.
Beverly Lean Out - Increases The Utilization Of Stored Fats For Energy!
Get Lean Out at Tiger Fitness
Increases The Utilization Of Stored Fats For Energy!
formula in capsule form providing the latest in quality substances including the standard lipotropics, L-Carnitine, Chromium Picolinate and Co-Enzyme 10, the substance that is an important fat soluble antioxidant which protects lipid-soluble substances like cholesterol from oxidation, the process where cholesterol is deposited on the arterial walls. This is the absolute optimum Lipotropic Formula in the world and should be in everyone's diet.
Burn Fat and Preserve Muscle
Have you ever dieted away your hard-earned muscle? What if you found a new substance that burned fat, enhanced fat utilization, spared muscle glycogen, and reduced muscle catabolism? You'd probably think you found a newly developed muscle building drug that the FDA doesn't know about. It's true that anabolic steroids certainly do all of the above. But, unlike steroids, your newfound substance does not raise blood serum triglycerides or lower HDL's (the "good" cholesterol). In fact, it does just the opposite giving you a much healthier blood lipid profile. If you saw both scientific and user reports suggesting that just such a substance exists, and the only reported side-effect was a euphoric mental state, would you be interested? Pretty ridiculous question, isn't it?
This is Legit
The fact is such a remarkable substance does exist. You probably even know a little about it. The name of this remarkable substance is L-Carnitine. Some of the fat burning products you've used in the past might list L-Carnitine on the label and you sure didn't experience the benefits noted above. That's okay, there are a number of reasons why that might have happened. Let's just say the scientific community has been impressed enough to fund a substantial amount of medical and scientific research centering around L-Carnitine. In fact, at least three U.S. pharmaceutical companies now make prescription tablets of L-Carnitine.
How it Works
L-Carnitine basically serves two extremely important functions as far as we're concerned as bodybuilders, burning fat and the metabolism of branched chain amino acids (BCAA's). L-Carnitine is a co-factor which is required for transport of fatty acids into the mitochondria of your muscle cells where they serve as a substrate for energy production while sparing muscle glycogen. Carnitine deficiency causes the fatty acids to back up and accumulate in the blood ultimately resulting in additional deposition of fat, usually right in the areas where you already have the greatest accumulation. L-Carnitine works synergistically with branched chain amino acids to increase their net metabolism. Remember that the BCAA's are the major amino acid constituents of your muscles. An increase in the amino acid content of the muscle will result in, at the very least, increased muscular hardness and possibly new muscle tissue. To sum up, L-Carnitine increases the utilization of fats for energy, sparing glycogen and amino acids. In essence, L-Carnitine shifts your fuel source utilization towards fats, leaving your amino acids and glycogen free to build muscle.
Important Facts Regarding L-Carnitine Supplementation
Research indicates the minimum dosage to minimize muscle catabolism while accelerating fat loss is 1000-1200 milligrams daily, divided into two doses. The dosage research proven most effective is 2-4 grams (2000-4000 mg) in 3-6 daily doses at three to six hour intervals. L-Carnitine is most effective taken on an empty stomach with water at least 45 minutes prior to eating and again 45 minutes prior to training. To maximize the effect of Carnitine on the metabolism of branch chained amino acids take 1200 mg L-Carnitine with a handful of BCAA's thirty minutes after training. You won't believe how quickly you'll recover. Cycle your L-Carnitine supplementation, 2-3 weeks on, 1-2 weeks off. L-Carnitine is non toxic but most people experience the fat loss benefits for only two or three weeks at a time due to the body's adaptation. We always use Lean Out, 4 per day, in the off season to help keep fats and cholesterol mobile in the body and to cleanse the liver. Starting at twelve weeks out we double the Lean Out dose to 8 per day. At eight weeks out we go up to 12 per day. Then at 4 - 5 weeks, we drop the Lean Out back to 4 per day and add in the Energy Reserve as indicated above.
EthiTech Nutrition - Yohimbine HCL - Kiss Those Trouble Areas GOODBYE!
Get it at Tiger Fitness
A lot of this information was learned from IFPA Pro Tommy Jeffers who was mentored by Lyle McDonald.
Yohimbe is harvested from trees grown in Africa and is used and sold as a supplement for fat loss and male sexuality. Yohimbine is a component of yohimbe and is the main alkaloid (and main active component) within yohimbe that produces the desired effects such as fat loss and male function (more shakin’ and bakin’).
We want the Yohimbine HCl—the ACTIVE EXTRACT-- not yohimbe bark which tends to not be accurately dosed. Thus, DO NOT BUY the bark, get the HCl version, as we make at EthiTech Nutrition. That way, we know what dose we are getting and can control the amount carefully, thus minimizing any side effects and maximizing results.
How Does Yohimbine HCl Work?
Yohimbine works on the alpha receptors. Alpha receptors inhibit fat breakdown, so wherever they are abundant, you have a harder time losing fat in that area. For example, they are in high concentration in men’s lower back and abs and in women’s hips and thighs. Yohimbine HCl acts as an alpha receptor blocker. Since alpha receptors inhibit fat breakdown, and Yohimbine HCl inhibits alpha receptors, the net result is that you get an INCREASE in fat breakdown. Thus, you get a significant increase in fat breakdown directly from an area that normally is the hardest place to lose it from. This is one reason why trainers like myself ALWAYS have my clients use this precontest!
Yohimbine HCL Dosing
Based on the studies we have examined, I recommend working up to 0.2mg/Kg of bodyweight. So that means a 220lb bodybuilder (divide by 2.2 to get Kg) is 100Kg and would work up to a dose of 20mg of Yohimbine HCl, or eight capsules of EthiTech Yohimbine HCl. I would typically start this person at one capsule and work up towards the full dose each consecutive session the person had no undesirable side effects.
As I was discussing with Joe Daniels of Swing This Kettlebells, Yohimbine HCl's effects work the best in the absence of insulin, and its effects start to diminish the more insulin is present. But this doesn’t mean that you cannot eat carbs! Insulin will usually be under control on a lower carb diet, like most trainers will put you on. Obviously, cardio first thing in the morning is a good time (with a scoop of MTS Nutrition Whey in your belly) and even post workout. What you can do is take the Yohimbine HCl DURING your weight training workout 15-20 minutes before you are done. Then, hop on the cardio post weights and BAM—FAT BLAST!
Exclusively at TigerFitness.com!
Some studies on YHCl we used to extrapolate this data:
1) Starke K, Trendelenburg AU, Limberger N. Presynaptic a 2-adrenoceptors: subtype determination. In: "Adrenoceptors: Structure, Function, and Pharmacology", Ruffolo, RR ed. Harwood Academic Publishers. 1995:99-108.
2) Byland, DB. Subtypes of a 1- and a 2-adrenergic receptors. FASEB J 1992; 6:832-839.
3) Heible JP, Ruffolo RR, Starke K. Identification, characterization and subclassification of a 2-adrenoceptors: an overview. In: "a 2-Adrenergic Receptors: Structure, Function and Therapeutic Implications". Lanier SM, Limbird LE eds. Harwood Academic Press. 1997:1-18.
4) Raiteri M, Bonanno G, Maura G, et al. Subclassification of release-regulating a 2-autorecptors in human breain cortex. Br J Pharmacol 1992; 107:1146-1151.
5) Piascik MT, Smith MS, Edelmann SE, et al. a 2 and a 1-Adrenergic receptors in the regulation of peripheral vascular function. In: "a 2-Adrenergic Receptors: Structure, Function and Therapeutic Implications". Lanier SM and Limbird LE eds. Harwood Academic Publishers. 1997:171-178.
6) Trendelenburg AU, Limberger N, Rump LC. a 2-Adrenergic receptors of the a 2C subtype mediate inhibition of norepinephrine release in human kidney cortex. Mol Pharmacol 1994; 415:M68-M76.
7) Galitzky J, Larrouy D, Berlan M, Lafontan M. New tools for human fat cell alpha2A-adrenoceptor characterization. Identification on membranes and on intact cells using the agonist [3H]RX821002. J Pharmacol Exp Ther 1990; 252:312-319.
8) Lafontan M, Berlan M. Fat cell alpha2-adrenoceptors: the regulation of fat cell function and lipolysis. Endocr Rev 1995 Dec, 16(6):716-738.
9) Arner P, Kriegholm E, et al. Adrenergic regulation of lipolysis in situ at rest and during exercise. J Clinical Invest 1990; 85:893-898.
10) Maurige P, J Galitzky, M Berlan, M Lafontan. Heterogeneous distribution of beta and alpha-2 adrenoceptor binding sites in human fat cells from various fat deposits: functional consequences. Eur J Clin Invest 1987; 17:156-165.
11) Lafontan M, et al. Adrenergic regulation of adipocyte metabolism. Hum Reprod 1997 Oct; 12 Suppl 1:6-20.
12) Gether U, Lin S, Kobilka BK. Delineating ligand-specific structural changes in adrenergic receptors by use of fluorescence spectroscopy. In: "a 2-Adrenergic Receptors: Structure, Function and Therapeutic Implications". Lanier SM and Limbird LE eds. 1997. Harwood Academic Publishers. 1997: 31-42.
13) Hodgetts V, Coppack S, Frayn KN, Hockaday TDR. Factors controlling fat mobilization from human subcutaneous adipose tissue during exercise. J Appl Phys 1991; 71:445-451.
14) Millet L, Barbe M, Lafontan M, Berlan M, Galitzky J. Catecholamine effects on lipolysis and blood flow in human abdominal and femoral adipose tissue. J Appl Physiol 1998; 85(1):181-188.
15) Ruffolo RR, Bondinell W, Hieble JP. a - and b -Adrenoceptors: From the gene to the clinic. 2. Structure-activity relationships and therapeutic applications. J Med Chem 1995; 38(19):3415-3444.
16) Arner P, Bolinder J. Microdialysis of adipose tissue. J Int Med 1991; 230:381-386.
17) Frayn KN, Fielding BA, Summers LKM. Investigation of human adipose tissue metabolism in vivo. J Endo 1997; 155:187-189.
18) Willette RN, Hieble JP, Sauermelch CF. The role of the alpha adrenoceptor subtypes in sympathetic control of the acralcutaneous microcirculation. J Pharmacol Exp Ther 1991; 256:599-605.
19) Borbujo J, Garcia-Villalon AL, Balle J, et al. Postjunctional alpha-1 and alpha-2 adrenoceptors in human skin arteries. An in vitro study. J Pharmacol Exp Ther 1989; 249:284-287.
20) Galitzky J, Lafontan M, Nordenstrom J, Arner P. Role of vascular alpha-2 adrenoceptors in regulating lipid mobilization from human adipose tissue. J Clin Invest 1993; 91:1997-2003.
21) Horn PT, Kohli JD, Listinsky JJ, Goldberg LI. Regional variation in the alpha-adrenergic receptors in the canine resistance vessels. Nauyn-Schmiedeberg’s Arch Pharmacol 1982; 318:166-172.
22) Flavahan NA, Cooke JP, Shepherd JT, Vanhoutte PM. Human postjunctional alpha-1 and alpha-2 adrenoceptors: differential distribution in arteries of the limbs. J Pharmacol Exp Ther 1987; 24:361-365.
23) Glusa E, Markwardt F. Characterization of postjunctional alpha-adrenoceptors in isolated human femoral veins and arteries. Nauyn-Schmiedeberg’s Arch Pharmacol 1983; 323:101-105.
24) Enoksson S, Nordenstrom J, Bolinder J, Arner P. Influence of local blood flow on glycerol levels in human adipose tissue. Int J Obesity 1995; 19:350-354.
25) Kovach AGB, Kovach E, Sandor P, et al. Metabolic responses to localized ischemia in adipose tissue. J Surg Res 1976; 20:37-44.
26) Barbe P, Galitzky J, Riviere D, Senard JM, et al. Effects of physiological and pharmaceutical variation of sympathetic nervous system on plasma non-esterified fatty acid concentrations in man. Br J Pharm 1993; 36:25-30.
27) Harmelen VV, Lonnqvist F, Thorne A, et al. Noradrenaline-induced lipolysis in isolated mesenteric, omental and subcutaneous adipocytes from obese subjects. Int J Obesity 1997; 21:972-979.
28) Arner P. Regulation of lipolysis in fat cells. Diab Rev 1996; 4:450-463.
29) Bjorntorp P. Obesity and the adipocyte. Neuroendocrine factors in obesity. J Endocrin 1997; 155:193-195.
30) Abate N, Garg A. Heterogeneity in adipose tissue metabolism: causes, implications, and management of regional adiposity. Prog Lipid Res 1995; 34:53-70.
31) Maurige P, et al. Regional difference in adipose tissue lipolysis from lean and obese women: existence of postreceptor alterations. Am J Physiol 1995 Aug; 269(2 pt 1): E341-E350.
32) Hellstrom L, Blaak E, Hagstrom-Toft E. Gender differences in adrenergic regulation of lipid mobilization during exercise. Int J Sports Med 1996; 17:439-447.
33) Goldberg MR Robertson D. Yohimbine: a pharmacological probe for study of the a 2-adrenoceptor. Pharmacol Rev 1983;35:143-180.
34) Berlan M, Galitzky J, Riviere D, et al. Plasma catecholamine levels and lipid mobilization induced by yohimbine in obese and non-obese women. Int J Obesity 1991; 15:305-315.
35) Galitzky J, Taouis M, Berlan M, Riviere D, et al. a 2-Antagonist compounds and lipid mobilization: evidence for a lipid mobilizing effect oral yohimbine in healthy male volunteers. Eur J Clin Invest 1988; 18:587-594.
36) Galitzky j, Riviere D, Tran MA, Montastruc JL, Berlan M. Pharmacodynamic effects of chronic yohimbine treatment in healthy volunteers. Eur J Clin Pharmacol 1990; 39:447-451.
37) Kuchio C, Jonderdo K, Piskorska D. Does yohimbine act as a slimming drug? Isr J Med Sci 1991; 27:550-556.
38) Zahorska-Markiewiz B, Kuchio, Piskorska D. Adrenergic control of lipolysis and metabolic responses in obesity. Horm Metabol Res. 1986; 18:693-697.
39) Murburg MM, Villacres EC, Ko BN, Veith RC. Effects of yohimbine on human sympathetic nervous system function. J Clin Endocrin Metab 1991; 73:861-865.
40) Sax L. Yohimbine does not affect fat distribution in men. Int J Obesity 1991; 3:261-280.
41) Bulow J. Adipose tissue blood flow during exercise. Dan Med Bull 1983; 30:85-100.
42) Bulow J, Madsen J. Regulation of fatty acid mobilization from adipose tissue during exercise. Scand J Sports Sci 1986; 8:19-26.
43) Bulow J, Madsen J, Astrup A, Christensen NJ. Vasoconstrictor effect of high FFA/albumin ratios in adipose tissue in vivo. Acta Physiol Scand 1985; 125:661-667.
44) Berlan M, Montastruc JL, Lafontan M. Pharmacological prospects for a 2-adrenoceptor antagonist therapy. TiPS Rev 1992; 13:277-282.
45) Montastruc P, Belan M, Monstastruc JL. Effect of yohimbine on submaxiallary salivation in dogs. Br. J Pharmac 1989; 98:101-104.
46) Chatelut E, Rispail Y, Berlan M, Montastruc JL. Yohimbine increases human salivary secretion. Br J Clin Pharmac 1989; 366-368.)
47) Fiaramonti J, Berlan M, Fargeas MJ, Bueno L. Yohimbine stimulates colonic motility through a central action in conscious dogs. J Gastrointes Mot 1992; 4:137-141.
48) Charney DS, Heninger GR, Breier A. Noradrenergic function in panic anxiety. Effects of yohimbine in healthy subjects and patients with agoraphobia and panic disorder. Arch Gen Psychiatry 1984; 41:751-763.
49) Murburg MM, Villacres EC, Ko BN, Veith RC. Effects of yohimbine on human sympathetic nervous system function. J Clin Endocrin Metab 1991; 73:861-865.
50) Grunhaus L, Tiongco D, Zelnik T, Flegel P, et al. Intravenous yohimbine. Selective enhancer of norepinephrine and cortisol secretion and systolic blood pressure in humans. Clin Neuropharmacol 1989; 12:106-114.
51) Henninger GR, Charney DS, Price LH. a 2-Adrenergic receptor sensitivity in depression. The plasma MHPG, behavioral and cardiovascular responses to yohimbine. Arc Gen Psychiatry 1988; 45:718-726.
52) McDougle CJ, Krystal JH, Price LH, Heninger GR, et al. Noradrenergic response to acute ethanol administration in healthy subjects: comparisons with intravenous yohimbine. Psychopharmacol 1995; 118:127-135.
53) Betz, JM, White KD. Gas chromatographic determination of yohimbine in commercial yohimbine products. J AOAC Int. 1995; 78:1189-1194.
54) Benedek IH, Blouin RA, McNamara PJ. Serum protein binding and the role of increased alpha1-acid glycoprotein in moderately obese subjects. Br J Clin Pharmacol 1984; 18:941-946.
55) Guthrie SK, Hariharan M, Grunhaus LJ. Yohimbine bioavailability in humans. Eur J Clin Pharmacol 1990; 39:409-411.
56) Hedner T, Edgar B, Edvinsson L, Hedner J, et al. Yohimbine pharmacokinetics and interaction with the sympathetic nervous system in normal volunteers. Eur J Clin Pharmacol 1992; 43:651-656.
57) Owen JA, Hakatsu SL, Fenemore J, Condra M, et al. The pharmacokinetics of yohimbine in man. Eur J Clin Pharmacol 1987; 32:577-582.
58) Le Verge R, Le Corre P, Chevanne F. Determination of yohimbine and its two hydroxylated metabolites in humans by high-performance liquid chromatography and mass spectral analysis. J Chromatog 1992; 574:283-292.
59) Brannan T, Martinez-Tica J, Yahr MD. Effect of yohimbine on brain monoamines: an in vivo study. J Neural Transm 1991; 3:81-87.
60) Hubbard JW, Pfister SL, Beidinger Am, Herzig TC, et al. The pharmacokinetic properties of yohimbine in the conscious rat. Naunyn-Schmiedeberg’s Arch Pharmacol 1988; 337:583-587.
The King of Fat Burners: OxyElite Pro - Is No Longer Available.
Get USP Labs OxyELITE Pro
Product Details
Listen, green tea has MANY benefits...It's an awesome antioxidant and is rightfully a staple in many intelligent lifters arsenals... And, yes, some science shows it boosts metabolism...but let's be honest, it's not gonna get you ripped. My grandmother's been drinking it every day for decades and I'm pretty sure she doesn't have a six-pack.
And synephrine...don't even get me started with that crap...It wasn't effective when it was initially touted as "the next ephedra" ...and it's not effective now - despite it blindly being included in pretty much every formula out there...
...I'm proud to say we've hit paydirt...
Introducing a burner so laser-targeted, it's been coined the "Super Thermogenic™" by researchers familiar with its effectiveness...
It's called OxyELITE Pro™ - Its potency is unmatched & its ability to deliver is flat-out staggering...
But don't believe me, check out what these lucky individuals who received a of this powerful thermogenic fat burner and appetite suppressant had to say...
OxyELITE Pro Reviews:
"The thermogenic effects were evident after about 10 minutes of dosing 1 capsule of OxyELITE Pro™ and continue, my whole body is sweating at the moment and it is about 68 in my place... Jacob, if I have to run my A/C in November!!!!"Matt Finnegan - Houston, TX"OxyELITE Pro™ takes away your appetite, I mean demolishes it. The only time I am actually hungry is in the morning when I first wake up. A big difference for me. Huge difference."Benjamin Johnston - Tampa, FL
"Definitely feeling the heat on this stuff. I'm staying warm throughout the day - I love that feeling! The appetite suppression is killer too! Forcing myself to eat today... Within about 10 minutes, I felt OxyELITE Pro™ kick in. I've used the ECA combo a few times in the past. This was like a walk down memory lane! I felt the rush of energy. I felt the heat, and I felt the loss of appetite kick in. I looked over at my sandwich while driving & laughed out loud!! I felt great!!"Daniel Edney - Baton Rouge, LA
"As a pharmacist myself, I'd approve it. On ingredients alone, OxyELITE Pro™ will outperform everything on the market or anything planning to come to market. Nothing touches what is being used/how they are being used. Just awesome. You have really outdone yourself Jacob!"Forum member MrBigPR
"I have gotten stronger while on OxyELITE Pro™ during my cut - which absolutely blew my mind, never thought it would happen. I never ran out of energy even on the days I went extremely low calorie. I have to say that it really helped me maintain my muscle mass as well. I have never been able to diet like I just did and maintain as much mass and strength as I have while using OxyELITE Pro™. Guys and gals, this is truly the thermogenic that everyone has been waiting for.. It certainly gets my recommendation and I'll never do a diet without it ever again."Sam Cox - Riley, Kansas
Why Is The Last Place To Lose Fat, The First Place To Gain Fat?
It just doesn't seem fair, but unfortunately it's true...It may be your stomach, your love handles or even your man boobs. Fat sticks to these areas like super glue and hangs on for dear life.
So why does this happen?
Your BodyFat is Being Held Hostage.It's called the Alpha-2 receptor – And, if fat loss is your goal - it's your absolute worst enemy...
If the Alpha-2 receptor wants you to store fat, you're going to store fat – and lots of it – in the worst possible places
The alpha-2 receptor is responsible for inhibiting lipolysis - which basically means it allows the body to store fat AND prevents the body from burning fat...
In other words, the Alpha-2 receptor is holding your fat hostage & refusing to let go!
The Alpha-2 Receptor is abundant in certain areas of your body & non-existent in others...
This is exactly why your biceps are lean, yet your stomach is as smooth as a baby's ass...
The Alpha-2 Receptor is the technical term for "trouble spots"...
When it's activated, these areas are in "lock-down" - unable to burn fat and storing it like a bear preparing for hibernation...
Full-Fledged Thermogenesis
Luckily, this is where the key ingredients in OxyELITE Pro™ shine...By utilizing a compound shown to block off & deactivate the Alpha-2 receptor, we can use this to our advantage to torch fat quickly & efficiently – exactly where you want fat loss to occur!
Think of the Key Ingredients in OxyELITE Pro™ as S.W.A.T – busting down doors and "freeing" your fat cells from their hostage situation...
So sit back & strap on your seatbelt as this very well may be the most important info you've ever read on fat loss...
The OxyELITE Pro™ Ingredients
Rauwolfia Canescens (Rauwolscine)
This particular compound has been shown to block off & deactivate the Alpha-2 receptor!It's related to yohimbine, a compound which is often included in various fat loss formulas because of its ability to antagonize alpha 2-adrenoceptors, allowing lipolysis itself, while also allowing lipolysis to continue from beta adrenoceptor agonism as well (11,12).
Here's the problem: While antagonizing alpha 2-adrenoceptors is great, yohimbine unfortunately also does the same for alpha 1-adrenoceptors – and this can actually hinder lipolysis (13).
So, on the other hand, while yohimbine helps to improve lipolysis by antagonizing alpha 2-adrenoceptors, it may also prevent lipolysis from occurring by antagonizing alpha 1-adrenoceptors...
Kind of defeats the purpose, huh? It's like robbing Peter to Pay Paul...
The end result is only a mediocre or small effect upon fat loss, or no detectable fat loss at all, which is what has been demonstrated in the literature with yohimbine supplementation (14-17).
Rauwolscine on the other hand, is just as potent at antagonizing alpha 2-adrenoceptors as yohimbine, yet it is 50 times less potent at alpha 1-adrenoceptors (18), making it much more selective and much less likely to reduce lipolysis by inhibiting alpha 1-adrenoceptor activity when compared to yohimbine.
vRauwolscine is clearly the better choice.
Last, beware of those products which contain the hydroxylated derivatives of yohimbine (e.g., 10-OH-yohimbine and 11-OH-yohimbine). They may look different but the truth is that they are no more effective than yohimbine (19).
This is simply a marketing gimmick designed to get the consumer to believe they are getting something truly unique and more effective.
Bauhinia purpurea L (Leaf & Pod [standardized for Bauhiniastatins 1-4])
This particular extract is very exciting because, due to various factors (e.g., stress, dieting, overtraining), many individuals may have impaired conversion of T4 to T3.We all know thyroid hormones play an important role in metabolic rate and an increase in thyroid hormone levels can produce significant increases in energy expenditure.
This is, after all, why bodybuilders and fitness athletes will use synthetic T3 as they know it can help dramatically increase the rate at which they reduce fat mass.
In fact, excess thyroid levels have been known to increase metabolic rate by 25-50% in some cases and a person that is hypothyroid can have a decrease in metabolic rate by as much as 40% below normal (20).
vNow, we're not saying that we can increase thyroid hormone levels to the same degree that you'd get from taking exogenous thyroid hormone, but imagine if you could get even a fraction of that – the results would speak for themselves!
An extract from the plant Bauhinia purpurea has been shown in healthy adult animal models to increase T4 by approximately 37% compared to controls, while increasing T3 by an amazing 83%! (21)
If this wasn't exciting enough, another study found that Bauhinia purpurea was able to completely restore the decrease in thyroid hormone levels seen when giving healthy adult animal models the drugs, dexamethasone (a potent glucocorticoid) and metformin (an anti-diabetic drug) (22).
Yet another reason that increasing thyroid hormone is so important is because it's known to sensitize or increase the lipolytic effects of adrenergic stimulation (i.e., through compounds such as the geranium and Cirsium oligophyllum extracts, along with caffeine) in fat cells (33).
Due to the results of the studies, the authors concluded that the plant is likely increasing the conversion of peripheral T4 to the more potent T3.
This is like adding a NOS injector to your ten speed!
Bacopa monnieri (Leaf)
This plant has also been shown in an animal model to naturally increase thyroid hormone production. In this case, the plant extract was shown to increase T4 levels by approximately 41% compared to controls (23).In this case, however, since there was no increase in T3 seen, the authors concluded that the plant extract directly stimulates the release of T4 from the thyroid gland, as opposed to Bauhinia purpurea, which increased the conversion of T4 to T3.
By combining the two plant extracts, the goal is to create a situation where your body will release more T4, while at the same time more of this newly released T4 will be converted to the more metabolically active T3.
This is truly the "best of both worlds" scenario & the end result potentially being a substantial increase in energy expenditure and decreased fat.
Cirsium Oligophyllum (Whole Plant Extract)
Our use of this plant is based upon an animal study which had shown that compared to a control group, the extract was able to reduce the gain of bodyweight and fat mass and specifically seemed to target subcutaneous fat mass over that of visceral fat mass with a ratio favoring the reduction of subcutaneous fat mass over that of visceral of nearly 9 fold!(5)Pharmacological studies revealed that the active constituent possesses beta adrenoceptor agonist activity (other beta adrenoceptor agonists include compounds like ephedrine and clenbuterol) and stimulates lipolysis in subcutaneous fat cells.
Yet another similarity that the Cirsium oligophyllum extract shares with beta adrenoceptor agonists is that it works synergistically with caffeine, producing a more powerful lipolytic effect that far exceeds the effect when used by itself (5).
In fact, the potency of caffeine & Cirsium Oligophyllum combined was nearly 10 times greater than Cirsium Oligophyllum by itself.
As a final note, Uncoupling Protein-1 expression was up-regulated 1.3 fold, relative to controls in those animals receiving Cirsium Oligophyllum topically; more on this later (5).
Caffeine
Caffeine is often noted for its ability to increase resting energy expenditure, while also increasing lipolysis and lipid oxidation (6-8)...In addition, it is noted for its ability to increase feelings of energy, alertness, concentration and euphoric effects (9,10).
Everyone throws caffeine in their formula...but few know why...we have a specific purpose on why we included it...
...Aside from the benefits that caffeine alone imparts, it can also provide synergistic effects when combined with other ingredients in OxyELITE Pro™...
In particular, the caffeine we have included may increase the effectiveness of the other fat loss agents included in the formula, including our Geranium extract as well as Cirsium oligophyllum.
...In fact, in the study performed with Cirsium oligophyllum, they found exactly that!
Caffeine was shown to provide a substantial increase in the effectiveness of the extract at promoting lipolysis in subcutaneous fat cells (5)...
Is Your Fat Burner Targeting The Wrong Fat?
Notice that we said subcutaneous fat cells as this is where many companies fail to make a distinction when creating fat loss products...Many compounds can produce a decrease in fat mass when it comes to visceral fat (i.e., the fat that surrounds your organs), but when it comes to improving how you look, subcutaneous fat is what you're concerned with as this is the fat that is just below your skin and smothers up your muscles...
How Much Subcutaneous Fat You Have Means The Difference Between Looking Shredded Or Looking Like You Own Stock In A Donut House.
Targeting visceral fat is important for overall health - and the compounds in OxyELITE Pro™ can target visceral fat too - but as you can see, targeting visceral fat alone isn't going to do much for that layer of fat surrounding your muscles...
You need to get rid of excess subcutaneous fat to make a lean physique a reality.
Geranium [Stems] (extracted for 1,3-Dimethylamylamine)
Geranium has been used for centuries as a food additive and part of certain cultures regular diets. One constitute of geranium is this compound - a sympathomimetic, aliphatic amine with indirect (i.e., by increasing norepinephrine levels) and/or direct activity at alpha and beta-adrenoceptors (1-3).This compound's influence upon the sympathetic nervous system (SAS) and norepinephrine levels is something very important to consider as the SAS and norepinephrine are considered to play important roles when it comes to influencing metabolic rate and especially the rate at which we burn calories while at rest (4).
What does this all of this mean? It means this compound can potentially help provide the stimulation needed for better workouts (i.e., a feeling of more energy and focus), while also directly stimulating the use of fat as fuel in your body.
The Final Frontier – The Incredible Fat Loss Pathway Science Had All But Given Up On!
Consider this the icing on the cake...the 10th Ring for Phil Jackson...the next Billion Bill Gates makes...Brown Adipose Tissue (BAT)
So, I'm sure we've all heard of this before, right?
It's often referred to as BAT, or simply as brown fat. It's very metabolically active and uses fat as fuel in order to produce heat. Pretty cool stuff, right?
Unfortunately, we were under the assumption we really only have significant amounts of brown fat when we're babies...
So, that's the end of the story, right? Well, very recently, the answer has become a solid, but surprising, no.
As it turns out, we fell into that trap often referred to as dogmatic thinking. If we're told something enough times and no one challenges it, we believe it...
Very recently, scientific studies have begun to call into question the notion adult humans lack BAT and that it has no potential to play any significant physiological role (24-29).
So, without any more procrastination, let's delve into the most recent findings:
- A recent study found that up to 96% of subjects evaluated have detectable levels of BAT! (24,25).
- It has been found that those that are overweight have lower BAT activity than those that are lean, something that isn't entirely a coincidence as having more BAT can potentially mean expending more calories and thus maintaining a lower weight (25).
- It has been estimated that more than half of all men and women likely have at least 10 grams of BAT in their body (26).
- BAT is so metabolically active that as little as 50 grams of BAT could potentially account for up to 20% of total daily energy expenditure of an adult human when maximally stimulated (25,26).
- In one subject that was shown to possess 63 grams of BAT, it was estimated that when fully activated, this amount of BAT could burn approximately 4.1 kilograms (around 9 lbs) of fat mass over a year (25,27).
- That's around 9 lbs of fat lost by doing nothing but sitting on your butt!
For example, let's start with a hypothetical 190 pound man with 12% bodyfat...
If he had 63 grams of BAT like the subject mentioned above & we can maximally stimulate BAT, he would lower his bodyfat down to 7.6%... by doing nothing differently except activating his BAT fat!
The difference in 12% & 7.6% bodyfat is flat-out staggering – 12% and people may ask you if you workout a few times a week...
A true 7.6% & you'll have chicks beggin' & dudes hatin'...
Ok Jacob, How Do We Recruit BAT?
The problem however is recruiting BAT and activating what little we have in the first place...
While exposure to cold is one method, it isn't exactly feasible to expose yourself to cold every day of the week...
And aside from that, it has been pointed out that various environmental changes for modern humans such as insulated buildings, clothing advances and heated transport systems have all reduced the need for thermogenesis via BAT and thus the amount of activated BAT has decreasedas well...
But all hope is not lost...
The other method is via sympathetic stimulation or in other words, by using direct and indirect-acting sympathomimetics (25,28).
Where OxyELITE Pro™ Goes From All-Time Great to First Ballot Hall of Fame
First, it's important to understand how BAT provides such benefits...Now, normal white adipose tissue or WAT (i.e., the stuff around your organs and smothering your muscles) is generally there to accept extra energy from calories ingested and accepts them for storage as triglycerides (fat).
We need this to insulate us from the cold and protect our internal organs. BAT, on the other hand doesn't store extra energy as it instead dissipates this extra energy in the form of heat.
How does it do this? A major factor is a protein called uncoupling protein-1 (UCP-1).
Protein Uncoupling
This protein is able to uncouple oxidative phosphorylation, allowing the energy from foodstuffs that we consume to be released as heat (burned) instead of stored as fat (25,28,29).Have you ever had one of those very lean friends that always complains about being, "hot" all of the time, even when it's the dead of winter?
These are the same friends that seem to eat nothing but junk food, yet still manage to stay lean all the time!
Well, BAT and UCP-1 activity may, in fact, be part of the reason!
Not The Same As DNP
It's important to note this type of Protein Uncoupling IS NOT the same as the dangerous, yet highly effective, uncoupling compounds such as DNP...Unlike DNP (an exogenous substance), UCPs are produced naturally in mammals, including humans...
Although they may have other roles, one is that they work to uncouple oxidative phosphorylation, hypothetically providing the same benefits as exogenous uncouplers (e.g., weight and fat loss), although this uncoupling is much more limited and won't lead to hyperthermia and the various side effects that exogenous and foreign uncouplers like DNP can lead to...
Granted, it also won't likely lead to the same weight loss and fat loss results as exogenous uncouplers like DNP, but it may help with fat loss through the same mechanism.
So, plants or compounds that up-regulate the expression of UCPs, such as this plant, may help with fat loss in this way.
How Key Ingredients in OxyELITE Pro™ May Attack BAT
Quick factoid: Did you know that about 14% of the increase in metabolism from ephedrine was attributed to BAT? (25)First, Geranium extract has sympathomimetic properties & this makes it a great candidate for increasing UCP-1 expression and recruiting or regaining BAT so that we may use it once again.
Secondly, Cirsium oligophyllum has beta adrenoceptor agonist activity and, as would be expected, has actually been shown to increase UCP-1 activity in BAT of an animal model (5).
Third, caffeine has been shown to increase norepinephrine by up to 75% in one study.
This increased norepinephrine again contributes to increasing UCP-1 activity in BAT, unfortunately, it also ends up activating alpha 2-adrenoceptors...
And by doing this, it actually inhibits some of the potential lipolytic and thermogenic activity of norepinephrine by inhibiting adenylyl cyclase (i.e., the enzyme which allows for cyclic adenosine monophosphate or cAMP formation and what ultimately leads to lipolysis) activity (25,28).
This is where rauwolscine comes in to play as it is an alpha 2-adrenoceptor antagonist. It prevents this inhibition of adenylyl cyclase and thus can potentially lead to a further increase in cAMP, lipolysis and thermogenesis via UCP-1...
Again, this is where rauwolscine shows its superiority to yohimbine...
You see, the alpha 1-adrenoceptor is also activated by norepinephrine and it also contributes to increased UCP-1 activity and subsequent thermogenesis (28,29,31,32).
At least one partial mechanism for how the alpha 1-adrenoceptor exerts such an effect is by increasing the conversion of T4 to T3 within BAT, which in turn increases UCP-1 expression.
So, although yohimbine has some beneficial effect upon fat loss, it's this blocking of activity at the alpha 1-adrenoceptor that becomes its downfall. Rauwolscine is 50 times less potent at antagonizing the alpha 1-adrenoceptor and thus makes a much better agent to use for fat loss.
Last, but certainly not least, we still have both of our plant extracts designed to increase the release of T4 (Bacopa monnieri) and increase the conversion of T4 to T3 (Bauhinia purpurea).
As we just mentioned, T3 plays a very important role in UCP-1 expression so if we can elevate levels further, it is beneficial for a number of reasons...
Of course, the fact that T3 can up-regulate beta adrenoceptor expression while down-regulating alpha 2-adrenoceptor expression, all while inhibiting phosphodiesterase (i.e., an enzyme that breaks down cAMP) activity further adds to increase or potentiate the effectiveness of the other ingredients (33).
References:
- Anonymous. New and nonofficial remedies: methylhexamine; forthane. J Am Med Assoc. 1950 Jul 29;143(13):1156
- Swanson EE and Chen KK. Comparison of pressor action of aliphatic amines. J Pharmacol Exp Ther. 1946 88(1):10-13
- Charlier R. Pharmacology of 2-amino-4-methylhexane. Private Trans. Arch Int Pharmacodyn Ther. 1950 Sep 1;83(4):573-584
- Diepvens K, Westerterp KR, Westerterp-Plantenga MS. Obesity and thermogenesis related to the consumption of caffeine, ephedrine, capsaicin, and green tea. Am J Physiol Regul Integr Comp Physiol. 2007 Jan;292(1):R77-85
- Mori S, Satou M, Kanazawa S, et al. Body fat mass reduction and up-regulation of uncoupling protein by novel lipolysis-promoting plant extract. Int J Biol Sci 2009;5(4):311-318
- Bracco D, Ferrarra JM, Arnaud MJ, et al. Effects of caffeine on energy metabolism, heart rate, and methylxanthine metabolism in lean and obese women. Am J Physiol. 1995 Oct;269(4 Pt 1):E671-E678
- Dullo AG, Geissler CA, Horton T, et al. Normal caffeine consumption: influence on thermogenesis and daily energy expenditure in lean and postobese human volunteers. Am J Clin Nutr. 1989 Jan;49(1):44-50
- Acheson KJ, Gremaud G, Meirim I, et al. Metabolic effects of caffeine in humans: lipid oxidation or futile cycling? Am J Clin Nutr. 2004 Jan;79(1):40-46
- Kaplan GB, Greenblatt DJ, Ehrenberg BL, et al. Dose-dependent pharmacokinetics and psychomotor effects of caffeine in humans. J Clin Pharmacol. 1997 Aug;37(8):693-703
- Smith A. Effects of caffeine on human behavior. Food Chem Toxicol. 2002 Sep;40(9):1243-1255
- Berlan M, Galitzky J, Riviere D, et al. Plasma catecholamine levels and lipid mobilization induced by yohimbine in obese and non-obese women. Int J Obes. 1991 May;15(5):305-315
- Galitzky J, Taouis M, Berlan M, et al. Alpha 2-antagonist compounds and lipid mobilization: evidence for a lipid mobilizing effect of oral yohimbine in healthy male volunteers. Eur J Clin Invest. 1988 Dec;18(6):587-594
- Flechtner-Mors M, Jenkinson CP, Alt A, et al. In vivo alpha(1)-adrenergic lipolytic activity in subcutaneous adipose tissue of obese subjects. J Pharmacol Exp Ther. 2002 Apr;301(1):229-233
- Sax L. Yohimbine does not affect fat distribution in men. Int J Obes. 1991 Sep;15(9):561-565
- Kucio C, Jonderko K, Piskorska D. Does yohimbine act as a slimming drug? Isr J Med Sci. 1991 Oct;27(10):550-556
- Berlin I, Stalla-Bourdillon A, Thuillier Y, et al. Lack of efficacy of yohimbine in the treatment of obesity. J Pharmacol. 1986 Jul-Sep;17(3):343-347
- Zahorska-Markiewicz B, Kucio C, et al. Adrenergic control of lipolysis and metabolic responses in obesity. Horm Metab Res. 1986 Oct;18(10):693-697
- Perry BD, U'Prichard DC. [3H]rauwolscine (alpha-yohimbine): a specific antagonist radioligand for brain alpha 2-adrenergic receptors. Eur J Pharmacol. 1981 Dec 17;76(4):461-464
- Berlan M, Le Verge R, Galitzky J, et al. Alpha 2-adrenoceptor antagonist potencies of two hydroxylated metabolites of yohimbine. Br J Pharmacol. 1993 Apr;108(4):927-932
- Kronenberg HM, Melmed S, Polonsky KS, Larsen PR.: Williams Textbook of Endocrinology, 11th ed., Saunders Elsevier, Philadelpha, PA, 2008.
- Panda S, Kar A. Withania somnifera and Bauhinia purpurea in the regulation of circulating thyroid hormone concentrations in female mice. J Ethnopharmacol. 1999 Nov 1;67(2):233-239
- Jatwa R, Kar A. Amelioration of metformin-induced hypothyroidism by Withania somnifera and Bauhinia purpurea extracts in Type 2 diabetic mice. Phytother Res. 2009 Aug;23(8):1140-1145
- Kar A, Panda S, Bharti S. Relative efficacy of three medicinal plant extracts in the alteration of thyroid hormone concentrations in male mice. J Ethnopharmacol. 2002 Jul;81(2):281-285
- van Marken Lichtenbelt WD, Vanhommerig JW, Smulders NM, et al. Cold-activated brown adipose tissue in healthy men. N Engl J Med. 2009 Apr 9;360(15):1500-1508
- Fruhbeck G, Becerril S, Sainz N, et al. BAT: a new target for human obesity? Trends Pharmacol Sci 2009 Aug;30(8):387-396
- Cypress AM, Lehman S, Williams G, et al. Identification and importance of brown adipose tissue in adult humans. N Engl J Med. 2009 Apr 9;360(15):1509-1517
- Virtanen KA, Lidell ME, Orava J, et al. Functional brown adipose tissue in healthy adults. N Engl J Med. 2009 Apr 9;360(15):1518-1525
- Cannon B, Nedergaard J. Brown adipose tissue: function and physiological significance. Physiol Rev. 2004 Jan;84(1):277-359
- Celi FS. Brown adipose tissue��"when it pays to be inefficient. N Engl J Med. 2009 Apr 9;360(15):1553-1556
- Robertson D, Frolich JC, Carr RK, et al. Effects of caffeine on plasma renin activity, catecholamines and blood pressure. N Engl J Med. 1978 Jan 26;298(4):181-186
- Silva JE, Larsen PR. Adrenergic activation of triiodothyronine production in brown adipose tissue. Nature. 1983 Oct 20-26;305(5936):712-713
- Raasmaja A, York DA. Pharmacological characterization of alpha1- and beta-adrenergic synergism of 5'DII activity in rat brown adipocytes. Arch Physiol Biochem. 2006 Feb;112(1):23-30
- Hellstrom L, Wahrenberg H, Reynisdottir S, et al. Catecholamine-induced adipocyte lipolysis in human hyperthyroidism. J Clin Endocrinol Metab. 1997 Jan;82(1):159-166
Need some new exercises to try in 2015 or a refresher on your favorites? Here are 184: http://t.co/5mqUD0AOo1 pic.twitter.com/pAiUSqHzdw
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01.01.2015 | New Year's Day fitness and strength challenge. Prove you want the podium http://t.co/3IRGccGVfR @USPlabs pic.twitter.com/jeGkyRL0cO
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Low-carbohydrate diet burns more excess liver fat than low-calorie diet.
Press: http://t.co/xX7NGG4eZ5
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— Michael Eades, M.D. (@DrEades) December 8, 2014
Friday, March 15, 2013
Thursday, March 14, 2013
Epiq Ripped Fat Burner
GREEN COFFEE EXTRACT
Green coffee extract is from unroasted
coffee beans allowing the natural chlorogenic acid
to remain. Chlorogenic acid works with the
caffeine in coffee beans to burn up fat freed by caffeine from fat stores.
CAFFEINE ANHYDROUS
Caffeine spurs fat loss by both freeing up more body
fat from your fat stores and by boost your metabolism.
YOHIMBE
Yohimbe binds to fat cells and inhibits receptors that limit the amount of fat that can leave the fat cells.
CAYENNE PEPPER FRUIT
Capsaicin
increases levels of the neurotransmitter/ hormone norepinephrine which in turn increases
metabolism.
RASPBERRY KETONES
Raspberry ketones activates the hormone-sensitive
enzyme lipase which prods fat stored in fat cells to be broken down and
released into the bloodstream to be burned for fuel.
Source Citation (MLA 7th
Edition)
Stoppani, Jim. "Epiq Ripped: an up-close look at a brand-new fat burner."
Joe Weider's Muscle & Fitness Feb. 2013: 60.
Wednesday, March 13, 2013
Life Extension Natural Glucose Absorption Control
| 60 vegetarian capsules Item Catalog Number: 01471 |
| This blend of four natural active ingredients is formulated to help support healthy blood glucose (blood sugar) levels in those already within normal range and metabolic wellness. It begins with iodine, a trace element involved in the production of T3 (triiodothyronine) and T4 (thyroxine), two thyroid hormones primarily responsible for regulating metabolism.10-12 Chromium is a critical micronutrient recognized for its vital role in maintaining healthy blood sugar levels in those already within normal range when used as part of a healthy diet.13-17 Phase 3™ Sucrase Inhibitor is an L-Arabinose and Chromium GlycoProtein Complex. Sucrase is an enzyme that breaks down sucrose to fructose and glucose in the digestive tract for absorption into the bloodstream. Phase 3™ contains a phytonutrient called L-Arabinose that affects sucrase digestion and works synergistically with chromium to support healthy blood sugar and insulin levels in those already within normal range.66 The Alpha-Amylase EnzymeAging reduces our ability to utilize the carbohydrates (and fats) that constitute what most would consider part of a healthy diet. The result is that as we grow older, our bloodstreams become chronically bloated with glucose and triglycerides in direct contrast to the youthful metabolic profile we have in our younger years. Emerging scientific research suggests that the alpha-amylase enzyme plays an undesirable role in the digestion of dietary carbohydrates and subsequent absorption of calories from starch and sugar.A natural bean extract (Phaseolus vulgaris) moderates alpha-amylase activity. In a human trial in which all overweight participants were placed on a 2,000–2,200-calorie, carbohydrate-rich diet, those taking Phaseolus vulgaris lost 6.5 pounds and 1.2 inches in waist size in only 30 days compared with 0.8 pounds and 0.2 inches in the placebo group.32 Optimized Irvingia contains the identical Phaseolus vulgaris used in this study. The Alpha-Glucosidase EnzymeAnother intestinal enzyme that enables carbohydrate absorption is alpha-glucosidase. A patented seaweed extract (InSea2®) has demonstrated the ability to help maintain healthy levels of both alpha-glucosidase and alpha-amylase. When given to laboratory animals, this seaweed extract reduced after-meal (postprandial) glucose elevations by up to 90% compared with non-supplemented animals.63 |
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