Bottom line: Let's get away from the "fructose vs. the rest of
the world" discussion and focus on those "foods" that contain
significant amounts of fructose. If you click on "foods highest in
fructose" on nutritiondata.com, you will obviously find "pops, sodas,
and soft drinks" on the first three ranks. And while they do have a high
fructose concentration (29.8g per 200ml serving), the study at hand
should remind you of another thing they have in common... ha? Yeah!
Right, they are liquid fast absorbing and a real stressor for your liver.
should now start blaming liquid foods for everything, but the results
Ritze et al. present in their latest paper do in fact "provide evidence
that liquid versus solid high-sugar diets differentially modulate
feeding behavior, distinct intestinal sugar transporters and weight
regulating hormones" and may thus be "a critical component for the
development of obesity and fatty liver disease", not just in mice, but
also in humans, where Ritze et al. found "similar enhanced sugar
transporter regulation within the small intestine as in liquid
high-sugar diet fed mice" and previous studies suggest that simply
replacing energy containing drinks with water will inhibit or at least
slow down long-term weight gain (Pan. 2013)
the world" discussion and focus on those "foods" that contain
significant amounts of fructose. If you click on "foods highest in
fructose" on nutritiondata.com, you will obviously find "pops, sodas,
and soft drinks" on the first three ranks. And while they do have a high
fructose concentration (29.8g per 200ml serving), the study at hand
should remind you of another thing they have in common... ha? Yeah!
Right, they are liquid fast absorbing and a real stressor for your liver.
should now start blaming liquid foods for everything, but the results
Ritze et al. present in their latest paper do in fact "provide evidence
that liquid versus solid high-sugar diets differentially modulate
feeding behavior, distinct intestinal sugar transporters and weight
regulating hormones" and may thus be "a critical component for the
development of obesity and fatty liver disease", not just in mice, but
also in humans, where Ritze et al. found "similar enhanced sugar
transporter regulation within the small intestine as in liquid
high-sugar diet fed mice" and previous studies suggest that simply
replacing energy containing drinks with water will inhibit or at least
slow down long-term weight gain (Pan. 2013)
- Bergheim, Ina, et al. "Antibiotics protect against fructose-induced
hepatic lipid accumulation in mice: role of endotoxin." Journal of
hepatology 48.6 (2008): 983-992. - Le Roux, C. W., et al. "Postprandial plasma ghrelin is suppressed
proportional to meal calorie content in normal-weight but not obese
subjects." The Journal of Clinical Endocrinology & Metabolism 90.2
(2005): 1068-1071. - Pan, An, et al. "Changes in water and beverage intake and long-term
weight changes: results from three prospective cohort studies."
International journal of obesity 37.10 (2013): 1378-1385. - Ritze, Yvonne, et al. "Effect of High Sugar Intake on Glucose
Transporter and Weight Regulating Hormones in Mice and Humans." PloS one
9.7 (2014): e101702.
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